Unfractionated heparin is associated with a lower rise of serum vascular cell adhesion molecule-1 in acute ischemic stroke patients (Q28193517)

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11 November 2019

title

Unfractionated heparin is associated with a lower rise of serum vascular cell adhesion molecule-1 in acute ischemic stroke patients (English)

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11 November 2019

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3

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11 November 2019

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John J Aponte

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11 November 2019

author name string

Angel Chamorro

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11 November 2019

Alvaro Cervera

2

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11 November 2019

Antonio Dávalos

4

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11 November 2019

Ana M Planas

6

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11 November 2019

language of work or name

English

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publication date

1 August 2002

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11 November 2019

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11 November 2019

volume

328

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11 November 2019

issue

3

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11 November 2019

page(s)

229-232

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Classification of subtype of acute ischemic stroke. Definitions for use in a multicenter clinical trial. TOAST. Trial of Org 10172 in Acute Stroke Treatment

11 November 2019

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Immediate anticoagulation in acute focal brain ischemia revisited: gathering the evidence

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Heparin in Acute Stroke With Atrial Fibrillation

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Polymorphonuclear leukocytes occlude capillaries following middle cerebral artery occlusion and reperfusion in baboons

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Astrocytes are the primary source of tissue factor in the murine central nervous system. A role for astrocytes in cerebral hemostasis

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Dichotomizing stroke outcomes based on self-reported dependency

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VLA-4 integrin cross-linking on human monocytic THP-1 cells induces tissue factor expression by a mechanism involving mitogen-activated protein kinase.

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Heparin induces release of extrinsic: Coagulation pathway inhibitor (EPI)

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Inflammation and glial responses in ischemic brain lesions.

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Early intrathecal production of interleukin-6 predicts the size of brain lesion in stroke.

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Heparin in inflammation: potential therapeutic applications beyond anticoagulation.

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Reduction of brain injury using heparin to inhibit leukocyte accumulation in a rat model of transient focal cerebral ischemia. II. Dose-response effect and the therapeutic window.

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7 January 2021

10.1016/S0304-3940(02)00518-9

Identifiers

DOI

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11 November 2019

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