Histochemically-reactive zinc in amyloid plaques, angiopathy, and degenerating neurons of Alzheimer's diseased brains (Q28144875)

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M S Jensen

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D S Silva

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P J Kesslak

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G Danscher

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C J Frederickson

Release of endogenous Zn2+ from brain tissue during activity.

7

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language of work or name

English

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publication date

10 January 2000

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published in

Brain Research

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volume

852

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page(s)

274-8

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issue

2

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cites work

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Rapid induction of Alzheimer A beta amyloid formation by zinc.

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Spontaneous and evoked release of endogenous Zn2+ in the hippocampal mossy fiber zone of the rat in situ.

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Histochemical demonstration of heavy metals. A revised version of the sulphide silver method suitable for both light and electronmicroscopy

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The dithizone, Timm's sulphide silver and the selenium methods demonstrate a chelatable pool of zinc in CNS. A proton activation (PIXE) analysis of carbon tetrachloride extracts from rat brains and spinal cords intravitally treated with dithizone.

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Increased amount of zinc in the hippocampus and amygdala of Alzheimer's diseased brains: a proton-induced X-ray emission spectroscopic analysis of cryostat sections from autopsy material.

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Autometallographic silver enhancement of zinc sulfide crystals created in cryostat sections from human brain biopsies: a new technique that makes it feasible to demonstrate zinc ions in tissue sections from biopsies and early autopsy material.

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The autometallographic zinc-sulphide method. A new approach involving in vivo creation of nanometer-sized zinc sulphide crystal lattices in zinc-enriched synaptic and secretory vesicles

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Translocation of zinc may contribute to seizure-induced death of neurons

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A quinoline fluorescence method for visualizing and assaying the histochemically reactive zinc (bouton zinc) in the brain

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Stable-isotope dilution measurement of zinc and lead in rat hippocampus and spinal cord.

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Distribution of histochemically reactive zinc in the forebrain of the rat

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Assembly of Alzheimer-like, insoluble filaments from brain cerebrosides.

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Stimulation-induced uptake and release of zinc in hippocampal slices

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The role of zinc in selective neuronal death after transient global cerebral ischemia

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Zn(2+) permeates Ca(2+) permeable AMPA/kainate channels and triggers selective neural injury

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inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/S0006-8993(99)02096-X

1 reference

Consolidated OpenCitations Corpus – April 2017

1367920

1367920

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference