Mrc1 transduces signals of DNA replication stress to activate Rad53. (Q27935521)

GOA release 2020-03-11

author name string

A A Alcasabas

1

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A J Osborn

2

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J Bachant

3

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F Hu

4

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P J Werler

5

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K Bousset

6

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K Furuya

7

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J F Diffley

8

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A M Carr

9

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S J Elledge

https://scigraph.springernature.com/pub.10.1038/ncb1101-958

10

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language of work or name

English

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publication date

November 2001

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1 reference

1 December 2016

3

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page(s)

958-65

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issue

11

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exact match

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cites work

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Regulation of RAD53 by the ATM-like kinases MEC1 and TEL1 in yeast cell cycle checkpoint pathways

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Spk1/Rad53 is regulated by Mec1-dependent protein phosphorylation in DNA replication and damage checkpoint pathways

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S-phase-specific activation of Cds1 kinase defines a subpathway of the checkpoint response in Schizosaccharomyces pombe

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Rad53 FHA domain associated with phosphorylated Rad9 in the DNA damage checkpoint

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Activation of Rad53 kinase in response to DNA damage and its effect in modulating phosphorylation of the lagging strand DNA polymerase.

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Damage and replication checkpoint control in fission yeast is ensured by interactions of Crb2, a protein with BRCT motif, with Cut5 and Chk1.

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Recovery from DNA replicational stress is the essential function of the S-phase checkpoint pathway

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Control of the DNA damage checkpoint by chk1 and rad53 protein kinases through distinct mechanisms

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Isolation and characterization of the Schizosaccharomyces pombe rhp9 gene: a gene required for the DNA damage checkpoint but not the replication checkpoint

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Claspin, a novel protein required for the activation of Chk1 during a DNA replication checkpoint response in Xenopus egg extracts

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The RAD9 gene controls the cell cycle response to DNA damage in Saccharomyces cerevisiae

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A checkpoint regulates the rate of progression through S phase in S. cerevisiae in response to DNA damage.

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Comprehensive identification of cell cycle-regulated genes of the yeast Saccharomyces cerevisiae by microarray hybridization

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A Mec1- and Rad53-dependent checkpoint controls late-firing origins of DNA replication

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The SAD1/RAD53 protein kinase controls multiple checkpoints and DNA damage-induced transcription in yeast

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The budding yeast Rad9 checkpoint protein is subjected to Mec1/Tel1-dependent hyperphosphorylation and interacts with Rad53 after DNA damage

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MEC1-dependent phosphorylation of Rad9p in response to DNA damage

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DNA damage checkpoints and DNA replication controls in Saccharomyces cerevisiae.

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Fission yeast wee1 protein kinase is not required for DMA damage-dependent mitotic arrest

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Fission yeast genes involved in coupling mitosis to completion of DNA replication

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Additional modules for versatile and economical PCR-based gene deletion and modification in Saccharomyces cerevisiae

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Mec1p is essential for phosphorylation of the yeast DNA damage checkpoint protein Ddc1p, which physically interacts with Mec3p.

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cdc25+ functions as an inducer in the mitotic control of fission yeast.

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Characterization of Schizosaccharomyces pombe Hus1: a PCNA-related protein that associates with Rad1 and Rad9

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Vectors and gene targeting modules for tandem affinity purification in Schizosaccharomyces pombe

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