Response to rituximab in patients with rheumatoid arthritis in different compartments of the immune system (Q27692640)

Rogier M. Thurlings

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2

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Danielle M. Gerlag

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3

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Koen Vos

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4

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publication date

November 2011

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published in

Arthritis & Rheumatology

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volume

63

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issue

11

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page(s)

3187-94

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Rituximab for rheumatoid arthritis refractory to anti-tumor necrosis factor therapy: Results of a multicenter, randomized, double-blind, placebo-controlled, phase III trial evaluating primary efficacy and safety at twenty-four weeks

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Highly sensitive B cell analysis predicts response to rituximab therapy in rheumatoid arthritis

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Serologic changes following B lymphocyte depletion therapy for rheumatoid arthritis

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Decrease in immunoglobulin free light chains in patients with rheumatoid arthritis upon rituximab (anti-CD20) treatment correlates with decrease in disease activity

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Steady-state generation of mucosal IgA+ plasmablasts is not abrogated by B-cell depletion therapy with rituximab

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Rituximab therapy reduces activated B cells in both the peripheral blood and bone marrow of patients with rheumatoid arthritis: depletion of memory B cells correlates with clinical response

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Synovial tissue sublining CD68 expression is a biomarker of therapeutic response in rheumatoid arthritis clinical trials: consistency across centers

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C5A anaphylatoxin and its seven transmembrane-segment receptor

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Measurement of the chemotactic complement fragment C5a in rheumatoid synovial fluids by radioimmunoassay: role of C5a in the acute inflammatory phase

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Complement and cellular cytotoxicity in antibody therapy of cancer

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Residual inflammation after rituximab treatment is associated with sustained synovial plasma cell infiltration and enhanced B cell repopulation

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Immunohistochemical analysis as a means to predict responsiveness to rituximab treatment

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21 January 2018

Identifiers

DOI

10.1002/ART.30567

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PubMed publication ID

21792840

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