CD38 exacerbates focal cytokine production, postischemic inflammation and brain injury after focal cerebral ischemia (Q27346383)

1

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Kerstin Lardong

2

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Mathias Gelderblom

3

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Peter Ludewig

4

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Frank Leypoldt

5

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Christian Gerloff

7

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Tim Magnus

Creative Commons Attribution 4.0 International

8

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language of work or name

English

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publication date

2011

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published in

PLOS One

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volume

6

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issue

5

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page(s)

e19046

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copyright license

start time

13 May 2011

1 reference

April 2022 Public Data File from Crossref

copyright status

copyrighted

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cites work

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Evolution and function of the ADP ribosyl cyclase/CD38 gene family in physiology and pathology

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Expression of monocyte chemoattractant protein-1 and macrophage inflammatory protein-1 after focal cerebral ischemia in the rat

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Absence of the chemokine receptor CCR2 protects against cerebral ischemia/reperfusion injury in mice.

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Regulatory T cells are key cerebroprotective immunomodulators in acute experimental stroke.

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Pivotal role of cerebral interleukin-17-producing gammadeltaT cells in the delayed phase of ischemic brain injury

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Nitroxyl exacerbates ischemic cerebral injury and oxidative neurotoxicity.

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CD38 is expressed selectively during the activation of a subset of mature T cells with reduced proliferation but improved potential to produce cytokines

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Differential and time-dependent expression of monocyte chemoattractant protein-1 mRNA by astrocytes and macrophages in rat brain: effects of ischemia and peripheral lipopolysaccharide administration.

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Monocyte chemoattractant protein-1 is increased in the cerebrospinal fluid of patients with ischemic stroke.

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Overexpression of monocyte chemoattractant protein 1 in the brain exacerbates ischemic brain injury and is associated with recruitment of inflammatory cells.

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Monocyte chemoattractant protein-1 messenger RNA expression in rat ischemic cortex

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PubMed

https://pubmed.ncbi.nlm.nih.gov/21625615

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Chemokines in ischemia and reperfusion

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/21625615

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1371/JOURNAL.PONE.0019046

1 reference

4282496

ADS bibcode

2011PLoSO...619046C

0 references

4282496

1 reference

1 reference

1 reference