Deficiency of FANCD2-associated nuclease KIAA1018/FAN1 sensitizes cells to interstrand crosslinking agents (Q24337605)

ubiquitin-dependent protein binding

GOA release 2020-03-11

1 reference

Fanconi-associated nuclease;Fanconi-associated nuclease 1 homolog CELE_C01G5.8

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

Alessandro A Sartori

8

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Michael Otmar Hengartner

object named as

Michael O Hengartner

9

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author name string

Katja Kratz

1

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Barbara Schöpf

2

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Svenja Kaden

3

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Ataman Sendoel

4

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Ralf Eberhard

5

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Claudio Lademann

6

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Elda Cannavó

7

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Josef Jiricny

Sister chromatid exchange

10

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language of work or name

English

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publication date

9 July 2010

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published in

Cell

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volume

142

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page(s)

77-88

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issue

1

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Mechanism of replication-coupled DNA interstrand crosslink repair

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The Fanconi anemia protein FANCM can promote branch migration of Holliday junctions and replication forks

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Interaction of the Fanconi anemia proteins and BRCA1 in a common pathway

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The BRIP1 helicase functions independently of BRCA1 in the Fanconi anemia pathway for DNA crosslink repair

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Emergence of a DNA-damage response network consisting of Fanconi anaemia and BRCA proteins

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The DNA crosslink-induced S-phase checkpoint depends on ATR-CHK1 and ATR-NBS1-FANCD2 pathways

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The structure-specific endonuclease Mus81-Eme1 promotes conversion of interstrand DNA crosslinks into double-strands breaks

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The FANCJ/MutLalpha interaction is required for correction of the cross-link response in FA-J cells

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Mismatch recognition and DNA-dependent stimulation of the ATPase activity of hMutSalpha is abolished by a single mutation in the hMSH6 subunit.

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Saccharomyces cerevisiae MutLalpha is a mismatch repair endonuclease

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DNA replication is required To elicit cellular responses to psoralen-induced DNA interstrand cross-links.

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hMutSbeta is required for the recognition and uncoupling of psoralen interstrand cross-links in vitro

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