Transcriptional activation of hypoxia-inducible factor-1alpha by HDAC4 and HDAC5 involves differential recruitment of p300 and FIH-1 (Q24324150)

Histone deacetylase 4

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positive regulation of DNA-binding transcription factor activity

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Hypoxia inducible factor 1, alpha subunit

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author name string

Hee-Won Seo

1

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Eun-Jin Kim

2

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Hyelin Na

3

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Mi-Ock Lee

Targeting HIF-1 for cancer therapy

4

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language of work or name

English

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publication date

5 January 2009

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published in

FEBS Letters

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volume

583

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issue

1

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page(s)

55-60

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cites work

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Signalling via the hypoxia-inducible factor-1alpha requires multiple posttranslational modifications

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Proline hydroxylation and gene expression

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PCAF is an HIF-1alpha cofactor that regulates p53 transcriptional activity in hypoxia.

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FIH-1: a novel protein that interacts with HIF-1alpha and VHL to mediate repression of HIF-1 transcriptional activity

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Asparagine hydroxylation of the HIF transactivation domain a hypoxic switch

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FIH-1 is an asparaginyl hydroxylase enzyme that regulates the transcriptional activity of hypoxia-inducible factor

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Histone deacetylases induce angiogenesis by negative regulation of tumor suppressor genes

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Metastasis-associated protein 1 enhances stability of hypoxia-inducible factor-1alpha protein by recruiting histone deacetylase 1.

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Class II histone deacetylases are associated with VHL-independent regulation of hypoxia-inducible factor 1 alpha

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MDM2-HDAC1-mediated deacetylation of p53 is required for its degradation

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Hepatitis B virus X protein enhances transcriptional activity of hypoxia-inducible factor-1alpha through activation of mitogen-activated protein kinase pathway

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Histone deacetylase 7 associates with hypoxia-inducible factor 1alpha and increases transcriptional activity

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C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation

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Posttranslational hydroxylation of ankyrin repeats in IkappaB proteins by the hypoxia-inducible factor (HIF) asparaginyl hydroxylase, factor inhibiting HIF (FIH)

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Histone deacetylase inhibitors and the promise of epigenetic (and more) treatments for cancer

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