Activation of microglia: a neuroinflammatory role for CAP37 (Q24323117)

Effect of bacterial wall lipopolysaccharide (LPS) on morphology, motility, and cytoskeletal organization of microglia in cultures

GOA release 2020-03-11

author name string

H Anne Pereira

series ordinal

1

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Xin Ruan

series ordinal

2

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Padmasini Kumar

series ordinal

3

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language of work or name

English

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publication date

January 2003

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published in

Glia

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volume

41

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issue

1

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page(s)

64-72

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cites work

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Nonsteroidal anti-inflammatory drug use and Alzheimer's disease: a case-control study in Rochester, Minnesota, 1980 through 1984.

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The role of anti-inflammatory drugs in the prevention and treatment of Alzheimer's disease

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Influence of lipoproteins on microglial degradation of Alzheimer's amyloid beta-protein

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Microglial contribution to oxidative stress in Alzheimer's disease.

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Inducible nitric oxide synthase activity of cloned murine microglial cells

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Microglia and cytokines in neurological disease, with special reference to AIDS and Alzheimer's disease

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Inflammation and Alzheimer's disease: relationships between pathogenic mechanisms and clinical expression.

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Covalent structure of two novel neutrophile leucocyte-derived proteins of porcine and human origin. Neutrophile elastase homologues with strong monocyte and fibroblast chemotactic activities

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Epidermal growth factor is a motility factor for microglial cells in vitro: evidence for EGF receptor expression.

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CAP 37, a 37 kD human neutrophil granule cationic protein shares homology with inflammatory proteinases

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CAP37, a human neutrophil-derived chemotactic factor with monocyte specific activity

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CAP37, a neutrophil granule-derived protein stimulates protein kinase C activity in endothelial cells

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Expression of CAP37, a novel inflammatory mediator, in Alzheimer's disease.

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Importance of immunological and inflammatory processes in the pathogenesis and therapy of Alzheimer's disease.

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