Modulation of the cardiac sodium channel Nav1.5 by fibroblast growth factor homologous factor 1B (Q24313401)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12401812%20AND%20SRC:MED&resulttype=core&format=json

14 December 2019

title

Modulation of the cardiac sodium channel Nav1.5 by fibroblast growth factor homologous factor 1B (English)

1 reference

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Sodium voltage-gated channel alpha subunit 5

14 December 2019

main subject

1 reference

Fibroblast growth factor 12

GOA release 2020-03-11

1 reference

fibroblast growth factor binding

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

author

Chuanju Liu

series ordinal

1

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14 December 2019

5

Stephen G Waxman

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14 December 2019

2

Sulayman D Dib-Hajj

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14 December 2019

4

Theodore R Cummins

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14 December 2019

author name string

Muthukrishnan Renganathan

series ordinal

3

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14 December 2019

language of work or name

English

0 references

publication date

24 October 2002

1 reference

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Journal of Biological Chemistry

14 December 2019

published in

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14 December 2019

volume

278

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14 December 2019

issue

2

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14 December 2019

page(s)

1029-1036

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From ionic currents to molecular mechanisms: the structure and function of voltage-gated sodium channels

14 December 2019

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Sodium channel beta1 and beta3 subunits associate with neurofascin through their extracellular immunoglobulin-like domain

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Direct interaction with contactin targets voltage-gated sodium channel Na(v)1.9/NaN to the cell membrane.

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Annexin II light chain regulates sensory neuron-specific sodium channel expression

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A calcium sensor in the sodium channel modulates cardiac excitability

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Isoform diversity among fibroblast growth factor homologous factors is generated by alternative promoter usage and differential splicing

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Fibroblast growth factor homologous factors are intracellular signaling proteins

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Fibroblast growth factor homologous factor 1B binds to the C terminus of the tetrodotoxin-resistant sodium channel rNav1.9a (NaN)

21 January 2018

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Mammalian Ras interacts directly with the serine/threonine kinase Raf

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Identification of a new family of tissue-specific basic helix-loop-helix proteins with a two-hybrid system

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The interferon-inducible nucleolar p204 protein binds the ribosomal RNA-specific UBF1 transcription factor and inhibits ribosomal RNA transcription

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The MyoD-inducible p204 protein overcomes the inhibition of myoblast differentiation by Id proteins

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Nitric oxide blocks fast, slow, and persistent Na+ channels in C-type DRG neurons by S-nitrosylation

21 January 2018

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Role of the C-terminal domain in inactivation of brain and cardiac sodium channels

21 January 2018

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Congenital long-QT syndrome caused by a novel mutation in a conserved acidic domain of the cardiac Na+ channel

21 January 2018

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Novel LQT-3 mutation affects Na+ channel activity through interactions between alpha- and beta1-subunits

21 January 2018

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Effects of flecainide in patients with new SCN5A mutation: mutation-specific therapy for long-QT syndrome?

21 January 2018

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21 January 2018

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21 January 2018

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Primary structure and functional expression of the human cardiac tetrodotoxin-insensitive voltage-dependent sodium channel

21 January 2018

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Molecular cloning of a putative tetrodotoxin-resistant rat heart Na+ channel isoform

21 January 2018

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Na(v)1.5 underlies the 'third TTX-R sodium current' in rat small DRG neurons

21 January 2018

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21 January 2018

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A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons

21 January 2018

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The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways

21 January 2018

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21 January 2018

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21 January 2018

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7 January 2021

Inherited Brugada and long QT-3 syndrome mutations of a single residue of the cardiac sodium channel confer distinct channel and clinical phenotypes

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Identifiers

DOI

10.1074/JBC.M207074200

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12401812%20AND%20SRC:MED&resulttype=core&format=json

14 December 2019

1 reference