LIGHT, a new member of the TNF superfamily, and lymphotoxin alpha are ligands for herpesvirus entry mediator (Q24308657)

2

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R I Montgomery

3

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K D Kochel

4

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T C Cheung

5

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G L Yu

6

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S Ruben

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M Murphy

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R J Eisenberg

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G H Cohen

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P G Spear

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C F Ware

Abnormal development of secondary lymphoid tissues in lymphotoxin beta-deficient mice

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language of work or name

English

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publication date

January 1998

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published in

Immunity

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volume

8

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page(s)

21-30

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issue

1

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Death effector domain-containing herpesvirus and poxvirus proteins inhibit both Fas- and TNFR1-induced apoptosis

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A metalloproteinase disintegrin that releases tumour-necrosis factor-alpha from cells

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Lymphotoxin beta, a novel member of the TNF family that forms a heteromeric complex with lymphotoxin on the cell surface

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Preparation and characterization of soluble recombinant heterotrimeric complexes of human lymphotoxins alpha and beta.

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Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas.

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Production of lymphotoxin (LT alpha) and a soluble dimeric form of its receptor using the baculovirus expression system.

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A lymphotoxin-beta-specific receptor

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Abnormal development of peripheral lymphoid organs in mice deficient in lymphotoxin

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Disrupted splenic architecture, but normal lymph node development in mice expressing a soluble lymphotoxin-beta receptor-IgG1 fusion protein.

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Lymphotoxin-alpha (LTalpha) supports development of splenic follicular structure that is required for IgG responses

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Aspartic acid 50 and tyrosine 108 are essential for receptor binding and cytotoxic activity of tumour necrosis factor beta (lymphotoxin)

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Cloning and expression of cDNA for human lymphotoxin, a lymphokine with tumour necrosis activity

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ATAR, a novel tumor necrosis factor receptor family member, signals through TRAF2 and TRAF5

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Herpes simplex virus glycoprotein D mediates interference with herpes simplex virus infection

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Structure of tumour necrosis factor

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On the key role of secondary lymphoid organs in antiviral immune responses studied in alymphoplastic (aly/aly) and spleenless (Hox11(-)/-) mutant mice.

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Distinct roles in lymphoid organogenesis for lymphotoxins alpha and beta revealed in lymphotoxin beta-deficient mice

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Chronic inflammation caused by lymphotoxin is lymphoid neogenesis

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A newly identified member of the tumor necrosis factor receptor superfamily with a wide tissue distribution and involvement in lymphocyte activation

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Molecular cloning and expression of the human 55 kd tumor necrosis factor receptor

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Herpesvirus entry mediator, a member of the tumor necrosis factor receptor (TNFR) family, interacts with members of the TNFR-associated factor family and activates the transcription factors NF-kappaB and AP-1

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Lymphotoxin-alpha-deficient and TNF receptor-I-deficient mice define developmental and functional characteristics of germinal centers

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Herpes simplex virus-1 entry into cells mediated by a novel member of the TNF/NGF receptor family

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The Epstein-Barr virus transforming protein LMP1 engages signaling proteins for the tumor necrosis factor receptor family

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Peyer's patch organogenesis is intact yet formation of B lymphocyte follicles is defective in peripheral lymphoid organs of mice deficient for tumor necrosis factor and its 55-kDa receptor

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Human tumour necrosis factor: precursor structure, expression and homology to lymphotoxin

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Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

1619295

1619295

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference