A brain-specific isoform of mitochondrial apoptosis-inducing factor: AIF2 (Q24297828)

M Bordi

3

0 references

Carsten Culmsee

C Culmsee

19

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Francesco Cecconi

F Cecconi

22

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Klas Blomgren

K Blomgren

21

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Guido Kroemer

13

F Harper

G Kroemer

0 references

Mauro Piacentini

12

M Piacentini

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author name string

E Hangen

1

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D De Zio

2

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C Zhu

4

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P Dessen

5

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F Caffin

6

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S Lachkar

7

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J-L Perfettini

8

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V Lazar

9

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J Benard

10

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G M Fimia

11

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G Pierron

14

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J M Vicencio

15

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A de Andrade

17

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G Höglinger

18

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P Rustin

20

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N Modjtahedi

Cell Death & Differentiation

24

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language of work or name

English

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publication date

July 2010

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published in

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volume

17

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issue

7

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page(s)

1155-66

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cites work

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The crystal structure of the mouse apoptosis-inducing factor AIF

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DNA binding is required for the apoptogenic action of apoptosis inducing factor

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AIF: not just an apoptosis-inducing factor

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Export of mitochondrial AIF in response to proapoptotic stimuli depends on processing at the intermembrane space

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The variability of the harlequin mouse phenotype resembles that of human mitochondrial-complex I-deficiency syndromes

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Redox-Linked Conformational Dynamics in Apoptosis-Inducing Factor

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Apoptosis-inducing factor: vital and lethal

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NADH oxidase activity of mitochondrial apoptosis-inducing factor

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AIF deficiency compromises oxidative phosphorylation

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The harlequin mouse mutation downregulates apoptosis-inducing factor

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Muscle-specific loss of apoptosis-inducing factor leads to mitochondrial dysfunction, skeletal muscle atrophy, and dilated cardiomyopathy

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Dissociating the dual roles of apoptosis-inducing factor in maintaining mitochondrial structure and apoptosis

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Loss of apoptosis-inducing factor results in cell-type-specific neurogenesis defects

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Downregulation of Apoptosis-Inducing Factor in Harlequin Mutant Mice Sensitizes the Myocardium to Oxidative Stress–Related Cell Death and Pressure Overload–Induced Decompensation

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Physical interaction of apoptosis-inducing factor with DNA and RNA.

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AIF and cyclophilin A cooperate in apoptosis-associated chromatinolysis

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Multiple pathways of cytochrome c release from mitochondria in apoptosis

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An increase in intracellular Ca2+ is required for the activation of mitochondrial calpain to release AIF during cell death

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Heat shock protein 70 binding inhibits the nuclear import of apoptosis-inducing factor.

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Cyclophilin A participates in the nuclear translocation of apoptosis-inducing factor in neurons after cerebral hypoxia-ischemia

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Mitochondrial membrane permeabilization in neuronal injury

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Deletion of the mitochondrial flavoprotein apoptosis inducing factor (AIF) induces beta-cell apoptosis and impairs beta-cell mass

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Dominant cell death induction by extramitochondrially targeted apoptosis-inducing factor

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AIFsh, a novel apoptosis-inducing factor (AIF) pro-apoptotic isoform with potential pathological relevance in human cancer.

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Identification and characterization of AIFsh2, a mitochondrial apoptosis-inducing factor (AIF) isoform with NADH oxidase activity.

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AIF suppresses chemical stress-induced apoptosis and maintains the transformed state of tumor cells

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Redox-dependent changes in molecular properties of mitochondrial apoptosis-inducing factor

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7 January 2021

Cysteine protease inhibition prevents mitochondrial apoptosis-inducing factor (AIF) release

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7 January 2021

Apoptosome inactivation rescues proneural and neural cells from neurodegeneration.

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Progressive degeneration of human mesencephalic neuron-derived cells triggered by dopamine-dependent oxidative stress is dependent on the mixed-lineage kinase pathway.

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