Activation of CAAT enhancer-binding protein delta (C/EBPdelta) by interleukin-1 negatively influences apolipoprotein C-III expression. (Q41091114)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9295296%20AND%20SRC:MED&resulttype=core&format=json

6 November 2019

title

Activation of CAAT enhancer-binding protein delta (C/EBPdelta) by interleukin-1 negatively influences apolipoprotein C-III expression (English)

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6 November 2019

6

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6 November 2019

Vassilis I Zannis

4

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6 November 2019

author name string

J M Lacorte

1

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6 November 2019

E Ktistaki

2

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6 November 2019

A Beigneux

3

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6 November 2019

J Chambaz

5

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6 November 2019

language of work or name

English

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publication date

1 September 1997

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Journal of Biological Chemistry

6 November 2019

number of pages

7

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based on heuristic

inferred from page(s)

published in

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6 November 2019

volume

272

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6 November 2019

page(s)

23578-23584

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6 November 2019

issue

38

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Hepatic acute phase reaction in vivo and in vitro

6 November 2019

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Serum amyloid A gene expression under acute-phase conditions involves participation of inducible C/EBP-beta and C/EBP-delta and their activation by phosphorylation

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Participation of the transcription factor C/EBP delta in the acute-phase regulation of the human gene for complement component C3

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Regulated expression of three C/EBP isoforms during adipose conversion of 3T3-L1 cells

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A member of the C/EBP family, NF-IL6 beta, forms a heterodimer and transcriptionally synergizes with NF-IL6

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Isolation of a recombinant copy of the gene encoding C/EBP

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IL-6DBP, a nuclear protein involved in interleukin-6 signal transduction, defines a new family of leucine zipper proteins related to C/EBP

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Distinct mechanisms for regulation of the interleukin-8 gene involve synergism and cooperativity between C/EBP and NF-kappa B

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Apo CIII gene transcription is regulated by a cytokine inducible NF-kappa B element

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Modulation of lipoprotein lipase activity by apolipoproteins. Effect of apolipoprotein C-III

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An apolipoprotein CIII haplotype protective against hypertriglyceridemia is specified by promoter and 3' untranslated region polymorphisms

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An indirect negative autoregulatory mechanism involved in hepatocyte nuclear factor-1 gene expression.

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