Depletion of canonical Wnt signaling components has a neuroprotective effect on midbrain dopaminergic neurons in an MPTP-induced mouse model of Parkinson's disease (Q28593498)

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Depletion of canonical Wnt signaling components has a neuroprotective effect on midbrain dopaminergic neurons in an MPTP-induced mouse model of Parkinson's disease
scientific journal article

    Statements

    title
    Depletion of canonical Wnt signaling components has a neuroprotective effect on midbrain dopaminergic neurons in an MPTP-induced mouse model of Parkinson's disease (English)
    1 reference
    stated in
    PubMed
    PubMed ID
    25009587
    retrieved
    31 January 2017
    author name string
    Ting-Li Dai
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    1
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    PubMed
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    25009587
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    31 January 2017
    Chan Zhang
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    2
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    25009587
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    31 January 2017
    Fang Peng
    series ordinal
    3
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    stated in
    PubMed
    PubMed ID
    25009587
    retrieved
    31 January 2017
    Xue-Yuan Niu
    series ordinal
    4
    1 reference
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    PubMed
    PubMed ID
    25009587
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    31 January 2017
    Ling Hu
    series ordinal
    5
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    stated in
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    PubMed ID
    25009587
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    31 January 2017
    Qiong Zhang
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    6
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    25009587
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    31 January 2017
    Ying Huang
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    7
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    25009587
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    31 January 2017
    Ling Chen
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    8
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    25009587
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    31 January 2017
    Lei Zhang
    series ordinal
    9
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    31 January 2017
    Weidong Zhu
    series ordinal
    10
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    25009587
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    31 January 2017
    Yu-Qiang Ding
    series ordinal
    11
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    25009587
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    31 January 2017
    Ning-Ning Song
    series ordinal
    12
    1 reference
    stated in
    PubMed
    PubMed ID
    25009587
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    31 January 2017
    Min Liao
    series ordinal
    13
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    stated in
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    25009587
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    31 January 2017
    publication date
    1 August 2014
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    25009587
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    31 January 2017
    volume
    8
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    stated in
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    PubMed ID
    25009587
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    31 January 2017
    issue
    2
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    stated in
    PubMed
    PubMed ID
    25009587
    retrieved
    31 January 2017
    page(s)
    384–390
    1 reference
    stated in
    PubMed
    PubMed ID
    25009587
    retrieved
    31 January 2017
    cites work

    Identifiers

    PMCID
    4079420
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