TRICK2, a new alternatively spliced receptor that transduces the cytotoxic signal from TRAIL (Q28248106)

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5 November 2019

title

TRICK2, a new alternatively spliced receptor that transduces the cytotoxic signal from TRAIL (English)

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5 November 2019

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1

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5 November 2019

author name string

J Mongkolsapaya

2

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5 November 2019

X N Xu

3

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5 November 2019

A E Cowper

4

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5 November 2019

A J McMichael

5

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5 November 2019

J I Bell

6

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5 November 2019

language of work or name

English

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publication date

1 September 1997

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5 November 2019

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5 November 2019

volume

7

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5 November 2019

issue

9

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5 November 2019

page(s)

693-696

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The receptor for the cytotoxic ligand TRAIL

5 November 2019

cites work

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LARD: a new lymphoid-specific death domain containing receptor regulated by alternative pre-mRNA splicing

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Signal transduction by DR3, a death domain-containing receptor related to TNFR-1 and CD95

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A death-domain-containing receptor that mediates apoptosis

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Apo-3, a new member of the tumor necrosis factor receptor family, contains a death domain and activates apoptosis and NF-kappa B

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TRAMP, a novel apoptosis-mediating receptor with sequence homology to tumor necrosis factor receptor 1 and Fas(Apo-1/CD95)

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Crystal structure of the soluble human 55 kd TNF receptor-human TNF beta complex: implications for TNF receptor activation

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Structures of the extracellular domain of the type I tumor necrosis factor receptor

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Activation of apoptosis by Apo-2 ligand is independent of FADD but blocked by CrmA.

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Self-association of the Death Domains of the p55 Tumor Necrosis Factor (TNF) Receptor and Fas/APO1 Prompts Signaling for TNF and Fas/APO1 Effects

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Induction of apoptosis by the mouse Nedd2 gene, which encodes a protein similar to the product of the Caenorhabditis elegans cell death gene ced-3 and the mammalian IL-1 beta-converting enzyme

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Molecular cloning and expression of the Fas ligand, a novel member of the tumor necrosis factor family

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Cytotoxic T cells deficient in both functional fas ligand and perforin show residual cytolytic activity yet lose their capacity to induce lethal acute graft-versus-host disease

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Evasion of cytotoxic T lymphocyte (CTL) responses by nef-dependent induction of Fas ligand (CD95L) expression on simian immunodeficiency virus-infected cells.

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Identifiers

DOI

2 references

Consolidated OpenCitations Corpus – April 2017

4887641

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9285725%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

4887641

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

2 references

4887641