Endothelial apoptosis in Braf-deficient mice (Q24314857)

animal organ morphogenesis

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

Braf transforming gene

1 reference

GOA release 2020-03-11

author name string

L Wojnowski

1

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A M Zimmer

2

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T W Beck

3

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H Hahn

4

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R Bernal

5

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U R Rapp

6

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A Zimmer

https://scigraph.springernature.com/pub.10.1038/ng0797-293

7

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language of work or name

English

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publication date

July 1997

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published in

Nature Genetics

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volume

16

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page(s)

293-7

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issue

3

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exact match

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cites work

Bcl-2 targets the protein kinase Raf-1 to mitochondria

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Mammalian Ras interacts directly with the serine/threonine kinase Raf

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Requisite role of angiopoietin-1, a ligand for the TIE2 receptor, during embryonic angiogenesis

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Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours

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Susceptibility to apoptosis is differentially regulated by c-myc and mutated Ha-ras oncogenes and is associated with endonuclease availability

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Rodent fibroblast tumours expressing human myc and ras genes: growth, metastasis and endogenous oncogene expression.

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Cellular commitment to oncogene-induced transformation or apoptosis is dependent on the transcription factor IRF-1.

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C. elegans lin-45 raf gene participates in let-60 ras-stimulated vulval differentiation.

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Post-natal lethality and neurological and gastrointestinal defects in mice with targeted disruption of the A-Raf protein kinase gene.

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The mouse B-raf gene encodes multiple protein isoforms with tissue-specific expression.

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Origins and assembly of avian embryonic blood vessels

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Differentiation of endothelium.

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The receptor tyrosine kinase TIE is required for integrity and survival of vascular endothelial cells.

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Detection and measurement of single-strand breaks in nuclear DNA in fixed lens sections

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Dominant-negative and targeted null mutations in the endothelial receptor tyrosine kinase, tek, reveal a critical role in vasculogenesis of the embryo

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Integrin alpha v beta 3 antagonists promote tumor regression by inducing apoptosis of angiogenic blood vessels

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Regulation of the Ras signalling pathway by protein-tyrosine kinases.

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Biochemical Analysis of MEK Activation in NIH3T3 Fibroblasts

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The mitogen-activated protein kinase cascade is activated by B-Raf in response to nerve growth factor through interaction with p21ras

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Conditionally oncogenic forms of the A-Raf and B-Raf protein kinases display different biological and biochemical properties in NIH 3T3 cells

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