Maintenance of endoplasmic reticulum (ER) homeostasis in herpes simplex virus type 1-infected cells through the association of a viral glycoprotein with PERK, a cellular ER stress sensor. (Q42634140)

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scientific article published on 17 January 2007

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English	Maintenance of endoplasmic reticulum (ER) homeostasis in herpes simplex virus type 1-infected cells through the association of a viral glycoprotein with PERK, a cellular ER stress sensor.	scientific article published on 17 January 2007

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scholarly article

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24 December 2019

Maintenance of endoplasmic reticulum (ER) homeostasis in herpes simplex virus type 1-infected cells through the association of a viral glycoprotein with PERK, a cellular ER stress sensor (English)

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24 December 2019

endoplasmic reticulum

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2

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24 December 2019

author name string

Matthew Mulvey

1

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24 December 2019

Ian Mohr

3

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publication date

17 January 2007

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number of pages

14

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Journal of Virology

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81

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24 December 2019

3377-3390

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7

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24 December 2019

Cotranslocational degradation protects the stressed endoplasmic reticulum from protein overload

1 reference

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Resistance of mRNA translation to acute endoplasmic reticulum stress-inducing agents in herpes simplex virus type 1-infected cells requires multiple virus-encoded functions.

1 reference

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Herpes simplex virus 1 infection activates the endoplasmic reticulum resident kinase PERK and mediates eIF-2alpha dephosphorylation by the gamma(1)34.5 protein

1 reference

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Regulation of eIF2alpha phosphorylation by different functions that act during discrete phases in the herpes simplex virus type 1 life cycle

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Protein synthesis and endoplasmic reticulum stress can be modulated by the hepatitis C virus envelope protein E2 through the eukaryotic initiation factor 2alpha kinase PERK

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Inhibition of PACT-mediated activation of PKR by the herpes simplex virus type 1 Us11 protein

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Replication of a cytopathic strain of bovine viral diarrhea virus activates PERK and induces endoplasmic reticulum stress-mediated apoptosis of MDBK cells

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1 reference

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12 December 2020

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10.1128/JVI.02191-06

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24 December 2019

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Europe PubMed Central

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24 December 2019

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24 December 2019

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