Carbon tetrachloride hepatotoxicity: an example of lethal cleavage (Q39899406)

15 September 2017

title

Carbon tetrachloride hepatotoxicity: an example of lethal cleavage (English)

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15 September 2017

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R O Rechnagel

1

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15 September 2017

E A Glende

series ordinal

2

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15 September 2017

publication date

1 November 1973

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CRC critical reviews in toxicology

15 September 2017

published in

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15 September 2017

volume

2

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15 September 2017

issue

3

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15 September 2017

page(s)

263-297

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publication type of scholarly work

15 September 2017

review article

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Reversal by adenine of the ethionine-induced lipid accumulation in the endoplasmic reticulum of the rat liver. A preliminary report

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The reaction of liver proteins with a metabolite of carbon tetrachloride

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Effect of pretreatment of rats with carbon tetrachloride on tolerance development

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Peroxidative Hemolysis of Red Blood Cells from Patients with Abetalipoproteinemia (Acanthocytosis)*

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Carbon tetrachloride-induced protection against carbon tetrachloride toxicity. The role of the liver microsomal drug-metabolizing system

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Lipid metabolites of carbon tetrachloride

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The phospholipid-dependence of UDP-glucuronyltransferase

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Biochemical effects of 1,1-dichloroethylene in rats: dissociation of its hepatotoxicity from a lipoperoxidative mechanism

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Comparison of the biochemical alterations elicited in livers from rats treated with carbon tetrachloride, chloroform, 1,1,2-trichloroethane and 1,1,1-trichloroethane

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Incorporation of radioactive- -aminolevulinic acid into microsomal cytochrome P 450 : selective breakdown of the hemoprotein by allylisopropylacetamide and carbon tetrachloride

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Properties of cytochrome P-450 as affected by environmental factors: qualitative changes due to administration of polycyclic hydrocarbons

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Studies of Paroxysmal Nocturnal Hemoglobinuria Erythrocytes: Increased Lysis and Lipid Peroxide Formation by Hydrogen Peroxide*

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The reactions of the sulfhydryl groups on the elongation factors Tu and Ts.

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An Enzyme-Based Theory of Obligate Anaerobiosis: The Physiological Function of Superoxide Dismutase

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The effect of diet and 1,1,1-trichloro-2,2-bis-(p-chlorophenyl)ethane (DDT) on microsomal hydroxylating enzymes and on sensitivity of rats to carbon tetrachloride poisoning

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Radioautographic comparison of the uptake of galactose-H and glucose-H3 in the golgi region of various cells secreting glycoproteins or mucopolysaccharides

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ON THE MECHANISM OF DRUG HYDROXYLATION IN RAT LIVER MICROSOMES.

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Croonian Lecture: Lethal Synthesis

21 January 2018

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Early onset of lipoperoxidation in rat liver after carbon tetrachloride administration

21 January 2018

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New data on the question of lipoperoxidation in carbon tetrachloride poisoning

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Alterations in microsomal electron transport, oxidative N-demethylation and azo-dye cleavage in carbon tetrachloride and dimethylnitrosamine-induced liver injury

21 January 2018

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21 January 2018

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21 January 2018

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Reversibility of liver damage in rats rendered resistant to carbon tetrachloride by prior carbon tetrachloride administration: Bearing on the lipoperoxidation hypothesis

21 January 2018

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21 January 2018

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21 January 2018

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Necrogenic Action of Carbon Tetrachloride in the Rat: A Speculative Mechanism Based on Activation

21 January 2018

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10.3109/10408447309082019

21 January 2018

Identifiers

DOI

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release_dethlqhqijagljh22xlkkuoztm

15 September 2017

Fatcat ID

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https://api.fatcat.wiki/v0/release/dethlqhqijagljh22xlkkuoztm

mapped directly with Wikidata item

24 November 2022

based on heuristic

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