A mitochondrial perspective on cell death (Q34134640)

31 July 2017

title

A mitochondrial perspective on cell death (English)

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31 July 2017

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Bernardi P

1

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31 July 2017

Petronilli V

series ordinal

2

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31 July 2017

Di Lisa F

series ordinal

3

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31 July 2017

Forte M

series ordinal

4

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31 July 2017

publication date

1 February 2001

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31 July 2017

number of pages

6

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Trends in Biochemical Sciences

inferred from page(s)

published in

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31 July 2017

volume

26

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31 July 2017

issue

2

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31 July 2017

page(s)

112-117

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publication type of scholarly work

31 July 2017

review article

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Regulation of mitochondrial metabolism by ER Ca2+ release: an intimate connection

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Mitochondrial function as a determinant of recovery or death in cell response to injury

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Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

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Apoptosis inducing factor (AIF): a phylogenetically old, caspase-independent effector of cell death

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Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

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Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins

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BCL-2 family members and the mitochondria in apoptosis

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Mitochondria and cell death. Mechanistic aspects and methodological issues

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Mitochondrial membrane potential and neuronal glutamate excitotoxicity: mortality and millivolts

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Keilin's Respiratory Chain Concept and Its Chemiosmotic Consequences

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Monitoring of relative mitochondrial membrane potential in living cells by fluorescence microscopy

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Simultaneous imaging of cell and mitochondrial membrane potentials

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Imaging in five dimensions: time-dependent membrane potentials in individual mitochondria

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7 January 2021

Quantitative assay by flow cytometry of the mitochondrial membrane potential in intact cells

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7 January 2021

Efficiency of P‐glycoprotein–Mediated Exclusion of Rhodamine Dyes from Multidrug‐Resistant Cells is Determined by their Passive Transmembrane Movement Rate

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Dicarbocyanine fluorescent probes of membrane potential block lymphocyte capping, deplete cellular ATP and inhibit respiration of isolated mitochondria

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Chloromethyltetramethylrosamine (Mitotracker Orange) induces the mitochondrial permeability transition and inhibits respiratory complex I. Implications for the mechanism of cytochrome c release.

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Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

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7 January 2021

Mitochondrial transport of cations: channels, exchangers, and permeability transition.

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7 January 2021

Mitochondrial control of cell death

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7 January 2021

Transient and long-lasting openings of the mitochondrial permeability transition pore can be monitored directly in intact cells by changes in mitochondrial calcein fluorescence

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7 January 2021

Commitment to apoptosis by GD3 ganglioside depends on opening of the mitochondrial permeability transition pore

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7 January 2021

Requirement for GD3 ganglioside in CD95- and ceramide-induced apoptosis.

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Direct interaction of GD3 ganglioside with mitochondria generates reactive oxygen species followed by mitochondrial permeability transition, cytochrome c release, and caspase activation

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7 January 2021

On the involvement of a cyclosporin A sensitive mitochondrial pore in myocardial reperfusion injury

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7 January 2021

Bcl-2 inhibits the mitochondrial release of an apoptogenic protease

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7 January 2021

The internal structure of mitochondria

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7 January 2021

Bcl-xL Regulates the Membrane Potential and Volume Homeostasis of Mitochondria

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7 January 2021

Preservation of mitochondrial structure and function after Bid- or Bax-mediated cytochrome c release

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7 January 2021

The coordinate release of cytochrome c during apoptosis is rapid, complete and kinetically invariant

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The Permeability Transition Pore Complex: A Target for Apoptosis Regulation by Caspases and Bcl-2–related Proteins

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Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

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7 January 2021

BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death

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7 January 2021

Electrophysiological study of a novel large pore formed by Bax and the voltage-dependent anion channel that is permeable to cytochrome c.

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7 January 2021

Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane

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7 January 2021

Bcl-xL prevents cell death following growth factor withdrawal by facilitating mitochondrial ATP/ADP exchange

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7 January 2021

Outer mitochondrial membrane permeability can regulate coupled respiration and cell survival

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7 January 2021

The pro-apoptotic proteins, Bid and Bax, cause a limited permeabilization of the mitochondrial outer membrane that is enhanced by cytosol

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7 January 2021

Requirement of JNK for stress-induced activation of the cytochrome c-mediated death pathway

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7 January 2021

Biochemical and genetic analysis of the mitochondrial response of yeast to BAX and BCL-X(L)

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Role of oxidative phosphorylation in Bax toxicity.

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7 January 2021

BNIP3 and genetic control of necrosis-like cell death through the mitochondrial permeability transition pore

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7 January 2021

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Identifiers

DOI

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31 July 2017

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