Overexpression of Helicard, a CARD-containing helicase cleaved during apoptosis, accelerates DNA degradation (Q34128741)

31 July 2017

title

Overexpression of Helicard, a CARD-containing helicase cleaved during apoptosis, accelerates DNA degradation (English)

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31 July 2017

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6

Jürg Tschopp

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31 July 2017

author name string

Magdalena Kovacsovics

1

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31 July 2017

Fabio Martinon

2

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31 July 2017

Olivier Micheau

3

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31 July 2017

Jean Luc Bodmer

4

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31 July 2017

Kay Hofmann

5

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31 July 2017

publication date

1 May 2002

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31 July 2017

language of work or name

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31 July 2017

volume

12

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31 July 2017

issue

10

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31 July 2017

page(s)

838-843

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Apoptotic DNA fragmentation

31 July 2017

cites work

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Cleavage of CAD inhibitor in CAD activation and DNA degradation during apoptosis

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Molecular characterization of mitochondrial apoptosis-inducing factor

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Endonuclease G is an apoptotic DNase when released from mitochondria

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Mitochondrial endonuclease G is important for apoptosis in C. elegans.

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DNA helicases in inherited human disorders

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Mechanisms for ATP-dependent chromatin remodelling

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The modular nature of apoptotic signaling proteins

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7 January 2021

The CARD domain: a new apoptotic signalling motif

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Cloning and characterization of a novel retinoid-inducible gene 1(RIG1) deriving from human gastric cancer cells

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Ded1p, a DEAD-box protein required for translation initiation in Saccharomyces cerevisiae, is an RNA helicase

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Mitochondria and apoptosis

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7 January 2021

Apoptosis by a cytosolic extract from Fas-activated cells

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p53-mediated apoptosis is attenuated in Werner syndrome cells

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A flexible motif search technique based on generalized profiles

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TRAMP, a novel apoptosis-mediating receptor with sequence homology to tumor necrosis factor receptor 1 and Fas(Apo-1/CD95)

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Equine herpesvirus-2 E10 gene product, but not its cellular homologue, activates NF-kappaB transcription factor and c-Jun N-terminal kinase

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Equine herpesvirus protein E10 induces membrane recruitment and phosphorylation of its cellular homologue, bcl-10

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10.1016/S0960-9822(02)00842-4

Identifiers

DOI

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31 July 2017

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