Cellular senescence: mitotic clock or culture shock? (Q34019131)

30 July 2017

title

Cellular senescence: mitotic clock or culture shock? (English)

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30 July 2017

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Sherr CJ

1

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30 July 2017

DePinho RA

series ordinal

2

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30 July 2017

publication date

1 August 2000

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30 July 2017

language of work or name

0 references

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30 July 2017

volume

102

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30 July 2017

issue

4

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30 July 2017

page(s)

407-410

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publication type of scholarly work

30 July 2017

review article

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Telomere shortening and tumor formation by mouse cells lacking telomerase RNA

cites work

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Extension of life-span by introduction of telomerase into normal human cells

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p16INK4A and p19ARF act in overlapping pathways in cellular immortalization

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p53 deficiency rescues the adverse effects of telomere loss and cooperates with telomere dysfunction to accelerate carcinogenesis

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Telomeres, telomerase and cancer

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The Hallmarks of Cancer

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The serial cultivation of human diploid cell strains

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Tumor suppression at the mouse INK4a locus mediated by the alternative reading frame product p19ARF

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p53- and ATM-dependent apoptosis induced by telomeres lacking TRF2

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Both Rb/p16INK4a inactivation and telomerase activity are required to immortalize human epithelial cells.

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Extended culture of mouse embryo cells without senescence: inhibition by serum

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Murine fibroblasts lacking p21 undergo senescence and are resistant to transformation by oncogenic Ras.

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Developmental and tissue-specific regulation of mouse telomerase and telomere length

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Evidence for a relationship between longevity of mammalian species and life spans of normal fibroblasts in vitro and erythrocytes in vivo

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Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

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Quantitative studies of the growth of mouse embryo cells in culture and their development into established lines

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atm and p53 cooperate in apoptosis and suppression of tumorigenesis, but not in resistance to acute radiation toxicity

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The two-stage mechanism controlling cellular senescence and immortalization

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Myc signaling via the ARF tumor suppressor regulates p53-dependent apoptosis and immortalization

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Cdkn2a, the cyclin-dependent kinase inhibitor encoding p16INK4aand p19ARF, is a candidate for the plasmacytoma susceptibility locus,Pctr1

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7 January 2021

10.1016/S0092-8674(00)00046-5

Identifiers

DOI

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30 July 2017

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