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Integrative analysis reveals selective 9p24.1 amplification, increased PD-1 ligand expression, and further induction via JAK2 in nodular sclerosing Hodgkin lymphoma and primary mediastinal large B-cell lymphoma. (Q33632062)

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English	Integrative analysis reveals selective 9p24.1 amplification, increased PD-1 ligand expression, and further induction via JAK2 in nodular sclerosing Hodgkin lymphoma and primary mediastinal large B-cell lymphoma.	scientific article

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7 February 2020

Integrative analysis reveals selective 9p24.1 amplification, increased PD-1 ligand expression, and further induction via JAK2 in nodular sclerosing Hodgkin lymphoma and primary mediastinal large B-cell lymphoma (English)

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7 February 2020

Hodgkin lymphoma

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7 February 2020

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7 February 2020

Przemysław Juszczyński

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Todd R Golub

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Donna Neuberg

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7 February 2020

Margaret A Shipp

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Margaret A Shipp

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Scott J Rodig

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Treeve Currie

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7 February 2020

Evan O'Donnell

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7 February 2020

Bjoern Chapuy

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7 February 2020

Kunihiko Takeyama

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7 February 2020

Jeffery L Kutok

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13 July 2010

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7 February 2020

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7 February 2020

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7 February 2020

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7 February 2020

3268-3277

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7 February 2020

Genome-wide copy number analysis of Hodgkin Reed-Sternberg cells identifies recurrent imbalances with correlations to treatment outcome

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Regulation of JAK2 by miR-135a: prognostic impact in classic Hodgkin lymphoma.

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The biology of Hodgkin's lymphoma

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Interferon regulatory factor-1 is prerequisite to the constitutive expression and IFN-gamma-induced upregulation of B7-H1 (CD274).

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Increased programmed death-1+ tumor-infiltrating lymphocytes in classical Hodgkin lymphoma substantiate reduced overall survival

1 reference

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Genomic imbalances including amplification of the tyrosine kinase gene JAK2 in CD30+ Hodgkin cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/20628145

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

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10.1182/BLOOD-2010-05-282780

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7 February 2020

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7 February 2020

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