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Paclitaxel-induced apoptosis in BJAB cells proceeds via a death receptor-independent, caspases-3/-8-driven mitochondrial amplification loop (Q33186742)

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scientific article published in April 2003

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English	Paclitaxel-induced apoptosis in BJAB cells proceeds via a death receptor-independent, caspases-3/-8-driven mitochondrial amplification loop	scientific article published in April 2003

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scholarly article

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Europe PubMed Central

PubMed publication ID

23 July 2017

Paclitaxel-induced apoptosis in BJAB cells proceeds via a death receptor-independent, caspases-3/-8-driven mitochondrial amplification loop (English)

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Europe PubMed Central

PubMed publication ID

23 July 2017

apoptotic process

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based on heuristic

inferred from title

Clarissa von Haefen

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object named as

Clarissa von Haefen

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Europe PubMed Central

PubMed publication ID

23 July 2017

author name string

Thomas Wieder

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23 July 2017

Frank Essmann

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23 July 2017

Klaus Schulze-Osthoff

4

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Europe PubMed Central

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23 July 2017

Bernd Dörken

5

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23 July 2017

Peter T Daniel

6

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23 July 2017

publication date

1 April 2003

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23 July 2017

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23 July 2017

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23 July 2017

2236-2247

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23 July 2017

Inhibition of drug-induced Fas ligand transcription and apoptosis by Bcl-XL

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Bcl-2 phosphorylation and proteasome-dependent degradation induced by paclitaxel treatment: consequences on sensitivity of isolated mitochondria to Bid.

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Damaged microtubules can inactivate BCL-2 by means of the mTOR kinase.

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Taxol-induced ceramide generation and apoptosis in human breast cancer cells

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Caspases: the executioners of apoptosis

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21 January 2018

Dissecting the pathways to death

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The kiss of death: promises and failures of death receptors and ligands in cancer therapy

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Taxol-induced cell cycle arrest and apoptosis: dose-response relationship in lung cancer cells of different wild-type p53 status and under isogenic condition

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The most unkindest cut of all: on the multiple roles of mammalian caspases

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Overexpression of caspase-3 restores sensitivity for drug-induced apoptosis in breast cancer cell lines with acquired drug resistance

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Sensitization for death receptor- or drug-induced apoptosis by re-expression of caspase-8 through demethylation or gene transfer

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Cell type specific involvement of death receptor and mitochondrial pathways in drug-induced apoptosis

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Inhibition of human caspases by peptide-based and macromolecular inhibitors

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Adenovirus-mediated overexpression of p14(ARF) induces p53 and Bax-independent apoptosis

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Death receptor recruitment of endogenous caspase-10 and apoptosis initiation in the absence of caspase-8

1 reference

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Involvement of CD95-independent caspase 8 activation in arsenic trioxide-induced apoptosis

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Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c.

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Membrane potential, anion and cation conductances in Ehrlich ascites tumor cells

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The role of caspases in development, immunity, and apoptotic signal transduction: lessons from knockout mice.

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Rapid Kinetics of tBid-induced Cytochrome c and Smac/DIABLO Release and Mitochondrial Depolarization

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FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

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An induced proximity model for caspase-8 activation.

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Ionizing radiation and chemotherapeutic drugs induce apoptosis in lymphocytes in the absence of Fas or FADD/MORT1 signaling. Implications for cancer therapy

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Caspase structure, proteolytic substrates, and function during apoptotic cell death

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2-Chloro-2'-deoxyadenosine induces apoptosis through the Fas/Fas ligand pathway in human leukemia cell line MOLT-4.

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A crucial role of caspase 3 and caspase 8 in paclitaxel-induced apoptosis

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Relapse in childhood acute lymphoblastic leukemia is associated with a decrease of the Bax/Bcl-2 ratio and loss of spontaneous caspase-3 processing in vivo

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The apoptosis promoting Bcl-2 homologues Bak and Nbk/Bik overcome drug resistance in Mdr-1-negative and Mdr-1-overexpressing breast cancer cell lines

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Mitochondrial membrane potential monitored by JC-1 dye.

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Bcl-2 oncoprotein protects the human prostatic carcinoma cell line PC3 from TRAIL-mediated apoptosis.

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Measurement of protein using bicinchoninic acid

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Staurosporine and conventional anticancer drugs induce overlapping, yet distinct pathways of apoptosis and caspase activation

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Activation of mitochondrial voltage-dependent anion channel by apro-apoptotic BH3-only protein Bim

1 reference

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21 January 2018

Intracellular mechanisms of TRAIL: apoptosis through mitochondrial-dependent and -independent pathways

1 reference

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Necessity of interleukin-1β converting enzyme cascade in taxotere-initiated death signaling

1 reference

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21 January 2018

Caspases: Enemies Within

1 reference

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21 January 2018

Ceramide induces mitochondrial activation and apoptosis via a Bax-dependent pathway in human carcinoma cells

1 reference

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21 January 2018

The effect of antimicrotubule agents on signal transduction pathways of apoptosis: a review

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Activation of caspase-8 in drug-induced apoptosis of B-lymphoid cells is independent of CD95/Fas receptor-ligand interaction and occurs downstream of caspase-3.

1 reference

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Bid acts on the permeability transition pore complex to induce apoptosis

1 reference

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Target protease specificity of the viral serpin CrmA. Analysis of five caspases

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Differential role of caspase-8 and BID activation during radiation- and CD95-induced apoptosis

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Sensitization of resistant lymphoma cells to irradiation-induced apoptosis by the death ligand TRAIL

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Caspase-8/FLICE functions as an executioner caspase in anticancer drug-induced apoptosis

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Serine protease inhibitor TPCK prevents Taxol-induced cell death and blocks c-Raf-1 and Bcl-2 phosphorylation in human breast carcinoma cells

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Cytochrome c release is upstream to activation of caspase-9, caspase-8, and caspase-3 in the enhanced apoptosis of anaplastic thyroid cancer cells induced by manumycin and paclitaxel

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10.1038/SJ.ONC.1206280

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Europe PubMed Central

PubMed publication ID

23 July 2017

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

23 July 2017

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