Degenerative disorders caused by Bcl-2 deficiency prevented by loss of its BH3-only antagonist Bim (Q28587387)

31 January 2017

title

Degenerative disorders caused by Bcl-2 deficiency prevented by loss of its BH3-only antagonist Bim (English)

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BCL2-like 11 (apoptosis facilitator)

31 January 2017

main subject

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B cell leukemia/lymphoma 2

GOA release 2020-03-11

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GOA release 2020-03-11

P. Bouillet

1

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Suzanne Cory

object named as

S. Cory

2

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Andreas Strasser

object named as

A. Strasser

4

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author name string

L. C. Zhang

3

1 reference

31 January 2017

J. M. Adams

5

1 reference

31 January 2017

publication date

1 November 2001

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31 January 2017

language of work or name

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31 January 2017

volume

1

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31 January 2017

issue

5

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31 January 2017

page(s)

645–653

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The Bcl-2 protein family: arbiters of cell survival

31 January 2017

cites work

1 reference

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7 January 2021

Life-or-death decisions by the Bcl-2 protein family.

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7 January 2021

Bcl-2 Rescues T Lymphopoiesis in Interleukin-7 Receptor–Deficient Mice

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7 January 2021

Bax is present as a high molecular weight oligomer/complex in the mitochondrial membrane of apoptotic cells

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7 January 2021

Proapoptotic Bcl-2 relative Bim required for certain apoptotic responses, leukocyte homeostasis, and to preclude autoimmunity

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7 January 2021

Gene structure alternative splicing, and chromosomal localization of pro-apoptotic Bcl-2 relative Bim

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7 January 2021

Translocation of C. elegans CED-4 to nuclear membranes during programmed cell death

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7 January 2021

BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis

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7 January 2021

The C. elegans Protein EGL-1 Is Required for Programmed Cell Death and Interacts with the Bcl-2–like Protein CED-9

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7 January 2021

BCL-2 family members and the mitochondria in apoptosis

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7 January 2021

BH3-Only proteins-essential initiators of apoptotic cell death

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7 January 2021

Apoptosis in metanephric development

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7 January 2021

Bcl-2 overexpression prevents apoptosis-induced Madin-Darby canine kidney simple epithelial cyst formation

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7 January 2021

The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues

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7 January 2021

Bclx regulates the survival of double-positive thymocytes

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7 January 2021

Bcl-2 can rescue T lymphocyte development in interleukin-7 receptor-deficient mice but not in mutant rag-1-/- mice

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7 January 2021

Bcl-2 prevents Bax oligomerization in the mitochondrial outer membrane

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7 January 2021

Bcl-2 inhibits Bax translocation from cytosol to mitochondria during drug-induced apoptosis of human tumor cells.

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7 January 2021

Targeted disruption of Bcl-2 alpha beta in mice: occurrence of gray hair, polycystic kidney disease, and lymphocytopenia

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7 January 2021

Bim: a novel member of the Bcl-2 family that promotes apoptosis

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7 January 2021

Demonstration of the in vivo interaction of key cell death regulators by structure-based design of second-site suppressors.

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7 January 2021

The biology of hair follicles

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7 January 2021

Induction of BIM, a proapoptotic BH3-only BCL-2 family member, is critical for neuronal apoptosis

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7 January 2021

Mouse models of cell death

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7 January 2021

A cre-transgenic mouse strain for the ubiquitous deletion of loxP-flanked gene segments including deletion in germ cells

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7 January 2021

bcl-2 transgene inhibits T cell death and perturbs thymic self-censorship

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7 January 2021

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7 January 2021

Bcl-2-deficient mice demonstrate fulminant lymphoid apoptosis, polycystic kidneys, and hypopigmented hair

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7 January 2021

Death by design: the big debut of small molecules

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7 January 2021

BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak

1 reference

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7 January 2021

10.1016/S1534-5807(01)00083-1

Identifiers

DOI

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Consolidated OpenCitations Corpus – April 2017

release_glvdlix7obhpllutbhi24hlvny

Fatcat ID

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Fatcat

https://api.fatcat.wiki/v0/release/glvdlix7obhpllutbhi24hlvny

24 November 2022

mapped directly with Wikidata item

1 reference

Consolidated OpenCitations Corpus – April 2017

1 reference

Consolidated OpenCitations Corpus – April 2017