Cytochrome c deficiency causes embryonic lethality and attenuates stress-induced apoptosis (Q28512752)

25 January 2017

title

Cytochrome c deficiency causes embryonic lethality and attenuates stress-induced apoptosis (English)

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Apoptotic peptidase activating factor 1

25 January 2017

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GOA release 2020-03-11

cytochrome c, somatic

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GOA release 2020-03-11

author name string

K. Li

1

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25 January 2017

Y. Li

2

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25 January 2017

J. M. Shelton

3

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25 January 2017

J. A. Richardson

4

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25 January 2017

E. Spencer

5

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25 January 2017

Z. J. Chen

6

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25 January 2017

X. Wang

7

1 reference

25 January 2017

R. S. Williams

8

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25 January 2017

publication date

12 May 2000

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25 January 2017

language of work or name

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25 January 2017

volume

101

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25 January 2017

issue

4

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25 January 2017

page(s)

389–399

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describes a project that uses

25 January 2017

Cyt c-/-

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Apaf1 (CED-4 homolog) regulates programmed cell death in mammalian development

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Mitochondria and apoptosis

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Differential requirement for caspase 9 in apoptotic pathways in vivo.

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Mitochondrial depolarization accompanies cytochrome c release during apoptosis in PC6 cells

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Bcl-2 blocks apoptosis in cells lacking mitochondrial DNA.

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Cytochrome c-mediated apoptosis in cells lacking mitochondrial DNA. Signaling pathway involving release and caspase 3 activation is conserved

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Cytochrome c activation of CPP32-like proteolysis plays a critical role in a Xenopus cell-free apoptosis system.

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Bid, a Bcl2 Interacting Protein, Mediates Cytochrome c Release from Mitochondria in Response to Activation of Cell Surface Death Receptors

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7 January 2021

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7 January 2021

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Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

release_7vapfw3h7ffpnjtkgim2nx3ele

Fatcat ID

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Fatcat

https://api.fatcat.wiki/v0/release/7vapfw3h7ffpnjtkgim2nx3ele

24 November 2022

mapped directly with Wikidata item

1 reference

Consolidated OpenCitations Corpus – April 2017

1 reference

Consolidated OpenCitations Corpus – April 2017