Mice lacking p35, a neuronal specific activator of Cdk5, display cortical lamination defects, seizures, and adult lethality (Q28302232)

GOA release 2020-03-11

author name string

T Chae

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Y T Kwon

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R Bronson

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P Dikkes

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E Li

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L H Tsai

Autoradiographic study of cell migration during histogenesis of cerebral cortex in the mouse

6

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publication date

January 1997

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language of work or name

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18

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issue

1

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page(s)

29-42

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cites work

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Temporal and spatial patterns of expression of p35, a regulatory subunit of cyclin-dependent kinase 5, in the nervous system of the mouse

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Two new mutants, ‘trembler’ and ‘reeler’, with neurological actions in the house mouse (Mus musculus L.)

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The mouse mutation reeler causes increased adhesion within a subpopulation of early postmitotic cortical neurons

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Neuronal cdc2-like kinase

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The cdk5/p35 kinase is essential for neurite outgrowth during neuronal differentiation

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The reeler gene-associated antigen on Cajal-Retzius neurons is a crucial molecule for laminar organization of cortical neurons.

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p35 is a neural-specific regulatory subunit of cyclin-dependent kinase 5

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