Oligomeric Bax is a component of the putative cytochrome c release channel MAC, mitochondrial apoptosis-induced channel (Q24523479)

1

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Sonia Martinez-Caballero

2

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Liang Guo

3

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Cynthia Hughes

4

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Oscar Teijido

5

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Thomas Ducret

6

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François Ichas

7

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Bruno Antonsson

9

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Elizabeth A Jonas

10

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Kathleen W Kinnally

Molecular Biology of the Cell

11

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publication date

May 2005

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language of work or name

English

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published in

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volume

16

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issue

5

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page(s)

2424-32

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cites work

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Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria

24 March 2017

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Conformation of the Bax C-terminus regulates subcellular location and cell death

24 March 2017

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Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

24 March 2017

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Inhibition of Bax channel-forming activity by Bcl-2

24 March 2017

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Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

24 March 2017

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Cell death: critical control points

24 March 2017

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BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis

24 March 2017

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Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

24 March 2017

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Bax in Murine Thymus Is a Soluble Monomeric Protein That Displays Differential Detergent-induced Conformations

24 March 2017

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VDAC2 inhibits BAK activation and mitochondrial apoptosis

24 March 2017

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Association of Bax and Bak homo-oligomers in mitochondria. Bax requirement for Bak reorganization and cytochrome c release

24 March 2017

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Bid, Bax, and lipids cooperate to form supramolecular openings in the outer mitochondrial membrane

24 March 2017

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Mitochondrial control of cell death

7 April 2017

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Laser micro-irradiation of mitochondria: is there an amplified mitochondrial death signal in neural cells?

7 April 2017

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The domains of apoptosis: a genomics perspective

27 September 2017

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Some amphiphilic cations block the mitochondrial apoptosis-induced channel, MAC.

27 September 2017

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Effects of cytochrome c on the mitochondrial apoptosis-induced channel MAC.

27 September 2017

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The Bcl-2 family: roles in cell survival and oncogenesis.

27 September 2017

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Selective disruption of lysosomes in HeLa cells triggers apoptosis mediated by cleavage of Bid by multiple papain-like lysosomal cathepsins

27 September 2017

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Inhibition of Bax-induced cytochrome c release from neural cell and brain mitochondria by dibucaine and propranolol.

27 September 2017

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The permeability transition pore signals apoptosis by directing Bax translocation and multimerization.

27 September 2017

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Bcl-2 prevents Bax oligomerization in the mitochondrial outer membrane

27 September 2017

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BAX-dependent transport of cytochrome c reconstituted in pure liposomes

27 September 2017

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Bcl-2 family proteins as ion-channels

27 September 2017

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Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis

27 September 2017

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Conversion of Bcl-2 to a Bax-like Death Effector by Caspases

27 September 2017

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Comparison of the ion channel characteristics of proapoptotic BAX and antiapoptotic BCL-2

27 September 2017

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Bcl-xL overexpression attenuates glutathione depletion in FL5.12 cells following interleukin-3 withdrawal

27 September 2017

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Channel formation by antiapoptotic protein Bcl-2.

27 September 2017

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Tim23, a protein import component of the mitochondrial inner membrane, is required for normal activity of the multiple conductance channel, MCC.

27 September 2017

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Structure of the outer mitochondrial membrane: ordered arrays of porelike subunits in outer-membrane fractions from neurospora crassa mitochondria

27 September 2017

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Response of yeast to the regulated expression of proteins in the Bcl-2 family

27 September 2017

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Why are mitochondria involved in apoptosis? Permeability transition pores and apoptosis as selective mechanisms to eliminate superoxide-producing mitochondria and cell

30 May 2018

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Lipidic pore formation by the concerted action of proapoptotic BAX and tBID.

30 May 2018

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Mitochondria and the Bcl-2 family proteins in apoptosis signaling pathways

29 November 2018

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BH3 death domain peptide induces cell type-selective mitochondrial outer membrane permeability

29 November 2018

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Bax is present as a high molecular weight oligomer/complex in the mitochondrial membrane of apoptotic cells

29 November 2018

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Expression and purification of full-length human Bax alpha

29 November 2018

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Purification and Biochemical Properties of Soluble Recombinant Human Bax

29 November 2018

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A membrane fusion strategy for single-channel recordings of membranes usually non-accessible to patch-clamp pipette electrodes

29 November 2018

1 reference

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Rapid spectrophotometric method for quantitation of cytochrome c release from isolated mitochondria or permeabilized cells revisited

29 November 2018

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12 December 2020

The mitochondrial apoptosis-induced channel (MAC) corresponds to a late apoptotic event

1 reference

12 December 2020

Ten years of publication in cell death

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12 December 2020

The C-terminus of bax is not a membrane addressing/anchoring signal

1 reference

12 December 2020