Induction of tyrosine hydroxylase expression by the transcription factor Pitx3 (Q24337808)

transcription, DNA-templated

GOA release 2020-03-11

1 reference

Paired-like homeodomain transcription factor 3

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

author name string

Kirsten Messmer

1

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Mary P Remington

2

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Frank Skidmore

3

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Paul S Fishman

International Journal of Developmental Neuroscience

4

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language of work or name

English

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publication date

February 2007

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published in

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volume

25

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page(s)

29-37

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issue

1

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cites work

Homeobox gene Pitx3 and its role in the development of dopamine neurons of the substantia nigra

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Nurr1, an orphan nuclear receptor with essential functions in developing dopamine cells

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5' upstream DNA sequence of the rat tyrosine hydroxylase gene directs high-level and tissue-specific expression to catecholaminergic neurons in the central nervous system of transgenic mice.

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The homeodomain transcription factor Pitx3 facilitates differentiation of mouse embryonic stem cells into AHD2-expressing dopaminergic neurons.

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The human tyrosine hydroxylase gene promoter

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Selective loss of dopaminergic neurons in the substantia nigra of Pitx3-deficient aphakia mice

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Dopamine neurons derived from embryonic stem cells function in an animal model of Parkinson's disease.

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In vitro expansion of hematopoietic stem cells by recombinant TAT-HOXB4 protein

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Ex vivo expansion of human hematopoietic stem cells by direct delivery of the HOXB4 homeoprotein

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Genetic engineering of mouse embryonic stem cells by Nurr1 enhances differentiation and maturation into dopaminergic neurons.

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A response element for the homeodomain transcription factor Ptx3 in the tyrosine hydroxylase gene promoter

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Pitx3 activates mouse tyrosine hydroxylase promoter via a high-affinity binding site

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Orphan nuclear receptor Nurr1 directly transactivates the promoter activity of the tyrosine hydroxylase gene in a cell-specific manner

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Dopaminergic neuronal differentiation from rat embryonic neural precursors by Nurr1 overexpression.

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Embryonic stem cells develop into functional dopaminergic neurons after transplantation in a Parkinson rat model

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Pitx3 is required for development of substantia nigra dopaminergic neurons.

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Cooperative transcription activation by Nurr1 and Pitx3 induces embryonic stem cell maturation to the midbrain dopamine neuron phenotype.

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A homeodomain gene Ptx3 has highly restricted brain expression in mesencephalic dopaminergic neurons

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Nurr1 is essential for the induction of the dopaminergic phenotype and the survival of ventral mesencephalic late dopaminergic precursor neurons

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Human tyrosine hydroxylase isoforms. Inhibition by excess tetrahydropterin and unusual behavior of isoform 3 after camp-dependent protein kinase phosphorylation

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Induction of cell cycle arrest and morphological differentiation by Nurr1 and retinoids in dopamine MN9D cells.

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Isolation of a new homeobox gene belonging to the Pitx/Rieg family: expression during lens development and mapping to the aphakia region on mouse chromosome 19.

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Deletion in the promoter region and altered expression of Pitx3 homeobox gene in aphakia mice.

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Transcription factors in the development of midbrain dopamine neurons

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Identifying combinatorial regulation of transcription factors and binding motifs

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Pitx3 is required for motor activity and for survival of a subset of midbrain dopaminergic neurons

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Early developmental failure of substantia nigra dopamine neurons in mice lacking the homeodomain gene Pitx3

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Development of the mesencephalic dopaminergic neuron system is compromised in the absence of neurogenin 2

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Lmx1b, Pet-1, and Nkx2.2 coordinately specify serotonergic neurotransmitter phenotype.

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