Tap: a novel cellular protein that interacts with tip of herpesvirus saimiri and induces lymphocyte aggregation (Q24336654)

2

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W Seol

3

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M DeMaria

4

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M Rosenzweig

5

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J U Jung

Primary structure of the herpesvirus saimiri genome

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language of work or name

English

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publication date

May 1997

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published in

Immunity

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volume

6

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page(s)

571-82

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issue

5

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cites work

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Herpesvirus saimiri has a gene specifying a homologue of the cellular membrane glycoprotein CD59

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An essential role for NF-kappaB in preventing TNF-alpha-induced cell death

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Phenotypic and functional consequences of herpesvirus saimiri infection of human CD8+ cytotoxic T lymphocytes.

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The divergence between two oncogenic Herpesvirus saimiri strains in a genomic region related to the transforming phenotype

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Stable growth transformation of human T lymphocytes by herpesvirus saimiri

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The product of the Herpesvirus saimiri open reading frame 1 (tip) interacts with T cell-specific kinase p56lck in transformed cells

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Identification of herpesvirus-like DNA sequences in AIDS-associated Kaposi's sarcoma

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A component of the 26S proteasome binds on orphan member of the nuclear hormone receptor superfamily

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A direct interaction between a glutamine-rich activator and the N terminus of TFIIB can mediate transcriptional activation in vivo

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Herpesvirus saimiri strain variability

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Cdi1, a human G1 and S phase protein phosphatase that associates with Cdk2

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Identification of transforming genes of subgroup A and C strains of Herpesvirus saimiri

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Identification of Lck-binding elements in tip of herpesvirus saimiri

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Downregulation of Lck-mediated signal transduction by tip of herpesvirus saimiri

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Molecular identification of I1PP2A, a novel potent heat-stable inhibitor protein of protein phosphatase 2A.

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The Epstein-Barr virus transforming protein LMP1 engages signaling proteins for the tumor necrosis factor receptor family

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Herpesvirus saimiri encodes homologues of G protein-coupled receptors and cyclins

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Epstein-Barr virus latent membrane protein (LMP1) and nuclear proteins 2 and 3C are effectors of phenotypic changes in B lymphocytes: EBNA-2 and LMP1 cooperatively induce CD23

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Herpesvirus Saimiri encodes a new cytokine, IL-17, which binds to a novel cytokine receptor

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