CFTR regulates outwardly rectifying chloride channels through an autocrine mechanism involving ATP (Q24307520)

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M E Egan

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T H Hwang

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S B Fulmer

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S S Allen

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G R Cutting

Non-invasive liposome-mediated gene delivery can correct the ion transport defect in cystic fibrosis mutant mice

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publication date

30 June 1995

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language of work or name

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81

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issue

7

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page(s)

1063-73

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cites work

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Chloride secretory response to extracellular ATP in human normal and cystic fibrosis nasal epithelia

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cAMP-activated Cl channels in CFTR-transfected cystic fibrosis pancreatic epithelial cells

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Defective regulation of outwardly rectifying Cl- channels by protein kinase A corrected by insertion of CFTR

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CFTR and outward rectifying chloride channels are distinct proteins with a regulatory relationship

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Extracellular ATP: effects, sources and fate

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Hyperabsorption of Na+ and raised Ca(2+)-mediated Cl- secretion in nasal epithelia of CF mice

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Cl- channels in CF: lack of activation by protein kinase C and cAMP-dependent protein kinase

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Correction of the ion transport defect in cystic fibrosis transgenic mice by gene therapy

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Normalization of raised sodium absorption and raised calcium-mediated chloride secretion by adenovirus-mediated expression of cystic fibrosis transmembrane conductance regulator in primary human cystic fibrosis airway epithelial cells

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Activation by extracellular nucleotides of chloride secretion in the airway epithelia of patients with cystic fibrosis

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An Immortalized Cystic Fibrosis Tracheal Epithelial Cell Line Homozygous for the ΔF508 CFTR Mutation

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Control of CFTR chloride conductance by ATP levels through non-hydrolytic binding

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Both CFTR and outwardly rectifying chloride channels contribute to cAMP-stimulated whole cell chloride currents

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Heterotrimeric G proteins, vesicle trafficking, and CFTR Cl- channels

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A Cystic Fibrosis Bronchial Epithelial Cell Line: Immortalization by Adeno-12-SV40 Infection

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