RIP: a novel protein containing a death domain that interacts with Fas/APO-1 (CD95) in yeast and causes cell death (Q24307376)

Receptor interacting serine/threonine kinase 1

GOA release 2020-03-11

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extrinsic apoptotic signaling pathway

GOA release 2020-03-11

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GOA release 2020-03-11

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B Z Stanger

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P Leder

2

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T H Lee

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E Kim

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B Seed

High efficiency transformation of intact yeast cells using single stranded nucleic acids as a carrier

5

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publication date

19 May 1995

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language of work or name

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81

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issue

4

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page(s)

513-23

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cites work

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DNA fragmentation and cell death is selectively triggered in activated human lymphocytes by Fas antigen engagement

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The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosis

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The p70 tumor necrosis factor receptor mediates cytotoxicity

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Human wee1 maintains mitotic timing by protecting the nucleus from cytoplasmically activated Cdc2 kinase

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Tumor necrosis factor's cytotoxic activity is signaled by the p55 TNF receptor

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A novel domain within the 55 kd TNF receptor signals cell death

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Mxi1, a protein that specifically interacts with Max to bind Myc-Max recognition sites

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The TNF receptor superfamily of cellular and viral proteins: activation, costimulation, and death

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Cloning and characterization of a novel member of protein kinase family from soybean

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Lymphocyte-mediated cytotoxicity: two pathways and multiple effector molecules

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Fatal interactions: Fas-induced apoptosis of mature T cells

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Two distinct pathways of specific killing revealed by perforin mutant cytotoxic T lymphocytes

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The fas antigen is involved in peripheral but not thymic deletion of T lymphocytes in T cell receptor transgenic mice

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Three protein kinase structures define a common motif

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Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

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Cytolytic T-cell cytotoxicity is mediated through perforin and Fas lytic pathways

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Self-association of the Death Domains of the p55 Tumor Necrosis Factor (TNF) Receptor and Fas/APO1 Prompts Signaling for TNF and Fas/APO1 Effects

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Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

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Activation interferes with the APO-1 pathway in mature human T cells

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Fas and perforin pathways as major mechanisms of T cell-mediated cytotoxicity

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